« Previous
Next »
European Journal of Cancer
Volume 46, Issue 1
, Pages 191-197
, January 2010
Expression of NF-κB and IκB proteins in skeletal muscle of gastric cancer patients
References
- . Novel treatments for cancer cachexia. Expert Opin Invest Drugs. 2007;16(8):1241–1253
- . Mechanisms of cancer cachexia. Physiol Rev. 2009;89(2):381–410
- . Increased protein degradation and decreased protein synthesis in skeletal muscle during cancer cachexia. Br J Cancer. 1993;67(4):680–685
- Ca(2+)-dependent proteolysis in muscle wasting. Int J Biochem Cell Biol. 2005;37(10):2134–2146
- . Mechanisms to explain wasting of muscle and fat in cancer cachexia. Curr Opin Support Palliat Care. 2007;1(4):293–298
- . Cleavage of caspases-1, -3, -6, -8 and -9 substrates by proteases in skeletal muscles from mice undergoing cancer cachexia. Br J Cancer. 2001;84(8):1135–1140
- Proteasome activities in the rectus abdominis muscle of young and older individuals. Biogerontology. 2008;9(4):261–268
- . Increased expression of proteasome subunits in skeletal muscle of cancer patients with weight loss. Int J Biochem Cell Biol. 2005;37(10):2196–2206
- Increased muscle proteasome activity correlates with disease severity in gastric cancer patients. Ann Surg. 2003;237(3):384–389
- Muscle myostatin signalling is enhanced in experimental cancer cachexia. Eur J Clin Invest. 2008;38(7):531–538
- The AP-1/CJUN signaling cascade is involved in muscle differentiation: implications in muscle wasting during cancer cachexia. FEBS Lett. 2006;580(2):691–696
- . Anti-interferon-gamma antibody treatment, growth of Lewis lung tumours in mice and tumour-associated cachexia. Eur J Cancer. 1991;27(2):182–187
- Effect of RNA oligonucleotide targeting Foxo-1 on muscle growth in normal and cancer cachexia mice. Cancer Gene Ther. 2007;14(12):945–952
- IKKbeta/NF-kappaB activation causes severe muscle wasting in mice. Cell. 2004;119(2):285–298
- Dystrophin glycoprotein complex dysfunction: a regulatory link between muscular dystrophy and cancer cachexia. Cancer Cell. 2005;8(5):421–432
- Intratumoral injection of oligonucleotides to the NF-kappaB binding site inhibits cachexia in a mouse tumor model. Gene Ther. 1999;6(1):91–97
- . NF-kappaB mediates proteolysis-inducing factor induced protein degradation and expression of the ubiquitin-proteasome system in skeletal muscle. Br J Cancer. 2005;92(4):711–721
- . Skeletal muscle myocytes undergo protein loss and reactive oxygen-mediated NF-kappaB activation in response to tumor necrosis factor alpha. Faseb J. 1998;12(10):871–880
- . NF-kappaB mediates the protein loss induced by TNF-alpha in differentiated skeletal muscle myotubes [in process citation]. Am J Physiol Regul Integr Comp Physiol. 2000;279(4):R1165–R1170
- . NF-kappaB-induced loss of MyoD messenger RNA: possible role in muscle decay and cachexia [see comments]. Science. 2000;289(5488):2363–2366
- Role for I{kappa}B{alpha}, but not c-Rel, in skeletal muscle atrophy. Am J Physiol Cell Physiol. 2007;292(1):C372–C382
- Activation of an alternative NF-kappaB pathway in skeletal muscle during disuse atrophy. Faseb J. 2002;16(6):529–538
- . Disruption of either the Nfkb1 or the Bcl3 gene inhibits skeletal muscle atrophy. J Clin Invest. 2004;114(10):1504–1511
- Cancer cachexia is regulated by selective targeting of skeletal muscle gene products. J Clin Invest. 2004;114(3):370–378
- . Mitochondria-associated apoptotic signalling in denervated rat skeletal muscle. J Physiol. 2005;565(565):309–323
- . Introduction to NF-kappaB: players, pathways, perspectives. Oncogene. 2006;25(51):6680–6684
- . The candidate proto-oncogene bcl-3 encodes a transcriptional coactivator that activates through NF-kappa B p50 homodimers. Genes Dev. 1993;7(7B):1354–1363
- The oncoprotein Bcl-3 directly transactivates through kappaB motifs via association with DNA-binding p50B homodimers. Cell. 1993;72(5):729–739
- Exchange of N-CoR corepressor and Tip60 coactivator complexes links gene expression by NF-kappaB and beta-amyloid precursor protein. Cell. 2002;110(1):55–67
- . NF-kappaB activation and iNOS upregulation in skeletal muscle of patients with COPD and low body weight. Thorax. 2004;59(6):483–487
- . Phosphorylation of NF-kappaB and IkappaB proteins: implications in cancer and inflammation. Trends Biochem Sci. 2005;30(1):43–52
- . Tumor necrosis factor-regulated biphasic activation of NF-kappa B is required for cytokine-induced loss of skeletal muscle gene products. J Biol Chem. 2003;278(4):2294–2303[epub 002 November 12]
- Increased muscle ubiquitin mRNA levels in gastric cancer patients. Am J Physiol Regul Integr Comp Physiol. 2001;280(5):R1518–R1523
- Serum tumour necrosis factor-alpha levels in cancer patients are discontinuous and correlate with weight loss. Eur J Clin Invest. 2000;30(12):1107–1112
- TNF-alpha increases ubiquitin-conjugating activity in skeletal muscle by up-regulating UbcH2/E220k. Faseb J. 2003;17(9):1048–1057
- TNF-alpha acts via p38 MAPK to stimulate expression of the ubiquitin ligase atrogin1/MAFbx in skeletal muscle. Faseb J. 2005;19(3):362–370
- . TNM classification of malignant tumours. 6th ed.. New York: Wiley-Liss; 2002;
PII: S0959-8049(09)00733-3
doi: 10.1016/j.ejca.2009.10.008
© 2009 Elsevier Ltd. All rights reserved.
« Previous
Next »
European Journal of Cancer
Volume 46, Issue 1
, Pages 191-197
, January 2010
